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This is Scientific American's 60-Second Science. I'm Karen Hopkin.This'll just take a minute.

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It's one of marijuana's most well-known side effects: the ravenous desire for food commonly called the munchies.

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But why does weed make chocolate, chips and, as the Harold and Kumar documentaries have shown, White Castle sliders so irresistible?

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Finally, science may have an answer.

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The urge to eat is controlled by complex circuits of neurons in the brain.

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Some of these nerve cells make us feel hungry, driving us to eat.

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Others cause us to feel sated, so we put down the Doritos bag and stop filling our faces.

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To figure out how marijuana might hijack this system, researchers exposed mice to a chemical that mimics the effect of the active ingredient in cannabis, THC, by binding to the brain's THC receptors.

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These doped-up rodents tend to keep noshing, even if they've already eaten their fill.

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But what's going on in their little mouse brains?

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Or paradoxically, the researchers found that the cannabinoid receptor turns on the neurons that normally make animals feel full.

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But what happens next is different from usual.

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When the "hey-I'm-full" neurons get triggered by the THC receptors, they wind up sending a "hey-I'm-still-hungry" signal that send us scrambling for the cupcakes.

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(This is your brain on drugs.)

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In addition to explaining 4 a.m.diner trips, the research, in the journal Nature, may be useful for addressing the medical condition of appetite loss. It commonly happen with cancer and depression for example.

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Knowing these details of neuronal activity could lead to better treatments for those patients who could really benefit from a case of the munchies.

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Thanks for the minute, for Scientific American's 60-Second Science. I'm Karen Hopkin.

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欢迎使用 KMF 精听精研

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This is Scientific American's 60-Second Science. I'm Karen Hopkin.This'll just take a minute.

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It's one of marijuana's most well-known side effects: the ravenous desire for food commonly called the munchies.

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But why does weed make chocolate, chips and, as the Harold and Kumar documentaries have shown, White Castle sliders so irresistible?

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Finally, science may have an answer.

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The urge to eat is controlled by complex circuits of neurons in the brain.

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Some of these nerve cells make us feel hungry, driving us to eat.

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Others cause us to feel sated, so we put down the Doritos bag and stop filling our faces.

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To figure out how marijuana might hijack this system, researchers exposed mice to a chemical that mimics the effect of the active ingredient in cannabis, THC, by binding to the brain's THC receptors.

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These doped-up rodents tend to keep noshing, even if they've already eaten their fill.

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But what's going on in their little mouse brains?

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Or paradoxically, the researchers found that the cannabinoid receptor turns on the neurons that normally make animals feel full.

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But what happens next is different from usual.

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When the "hey-I'm-full" neurons get triggered by the THC receptors, they wind up sending a "hey-I'm-still-hungry" signal that send us scrambling for the cupcakes.

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(This is your brain on drugs.)

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In addition to explaining 4 a.m.diner trips, the research, in the journal Nature, may be useful for addressing the medical condition of appetite loss. It commonly happen with cancer and depression for example.

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Knowing these details of neuronal activity could lead to better treatments for those patients who could really benefit from a case of the munchies.

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Thanks for the minute, for Scientific American's 60-Second Science. I'm Karen Hopkin.

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